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Budoff June 2011

Member Forum >> UnKnown >> Budoff June 2011

CADHEART

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Posted: 10/26/2011 11:05:36 PM
 

Letter to the Editor

Assessment of progression of coronary atherosclerosis using multidetector computed tomography angiography (mdct)
 

Yasmin S. HamiraniCorresponding Author Contact Information, a, E-mail The Corresponding Author, Jigar Kadakiaa, Sandeep R. Pagalia, Irfan Zeba, Hussain Isma'eela, Naser Ahmadia, Guilda Sarrafa, TaeYoung Choia, Amish Patela, Matthew J. Budoffa

 

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Fig. 1.

Analyzing plaque in each segment using Sure Plaque program in vitals workstation (Vitrea) A) Selection of a vessel. B) Manual selection of segment with plaque with the automated program. C) Luminal and cross sectional views of selected segment with plaque. D) Demonstration of total, mixed, noncalcified and calcified plaque by the automated program based on preset HU [right hand corner of picture with red (noncalcified), blue (mixed) and yellow (calcified) colors]. (For interpretation of the references to color in this figure legend, the reader is referred to the web version of this article.)

 



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Fig. 2.

Change in total, noncalcified, mixed and calcified plaque on the repeat CT scan (y axis: annualized percentage change in plaque types). Tp: total plaque, sp: non-calcified plaque, mp: mixed plaque, cp: calcified plaque.

 



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Fig. 3.

Bland Altman correlation between two observers for calcified plaque (A), mixed plaque (B) and noncalcified plaque (C) volumes (mm3). Average of non-calcified plaque observer 1 and 2.

 


Table 1. General characteristics of patient population.
 
View table in article

Table 2. Coronary artery calcium and plaque quantification results on two MDCT scans.
 
View table in article

Table 3. Mean annualized percentage change in CAC and plaque volume.
 
View table in articleAPC: Annualized percentage change, CAC: coronary artery calcium score (AU-Agatson Units).

Table 4. Characteristics of patients with plaque progression on the follow up scan vs.non progressors.
 
View table in article(Progressors = annualized plaque change on MDCT > 0 mm3; Non-progressors = annualized plaque change on MDCT < 0 mm3).

Table 5. Differences in plaque types in progressors and non-progressor group.
 
View table in article

Table 6. Interobserver variability data on 30 segments for plaque measurement.
 
View table in article


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Posted: 10/26/2011 11:44:51 PM
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Posted: 10/26/2011 11:56:51 PM
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Posted: 10/27/2011 12:05:05 AM
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Posted: 10/27/2011 1:58:43 AM
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Posted: 10/27/2011 2:09:34 AM
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"Fair use" is not a copyright violation. and certainly not a criminal offense.   Sharing with others for educational purposes is fine and not a violation of any law.



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Posted: 10/27/2011 1:34:52 PM
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Posted: 10/27/2011 6:16:34 PM
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Posted: 10/28/2011 11:25:04 AM
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Posted: 10/28/2011 1:46:22 PM
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Posted: 10/28/2011 10:00:46 PM
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Posted: 10/29/2011 2:05:29 PM
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Posted: 10/29/2011 3:41:42 PM
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Posted: 10/30/2011 4:28:47 AM
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Posted: 10/30/2011 11:04:48 AM
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Posted: 10/31/2011 11:20:48 AM
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Posted: 2/16/2012 4:26:15 PM
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Posted: 5/23/2013 9:55:32 AM
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Table 3 chart above. 62 cohort. 1048 agatstons. 3.4% CAC annualized__________ How much EPA+DHA did coHort take!________the 62 coHort had varying Cardologists, Lipidologists


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Posted: 5/23/2013 10:04:25 AM
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Table3. Total Plaque. Scan1 to Scan2. 434 days between scans. .. Total coronary plaque (mm3) 787.7 ± 584.4 versus 771 ± 562.4____________How was this determined. In the setting of CAC and CTA
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Posted: 5/23/2013 10:13:12 AM
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Note Bunny's LipoProteins posted just above. ApoB 68. Hugely Larger diameters'D LDL's. What TYP'Rs would want those results? With very high amounts of Large diameteref HDL's.____________________ apoB 68 mg/dl by Berkeley Heart Lab!


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Posted: 5/26/2013 7:15:53 PM
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Strim Steve. 62 coHort here OBVIOUSLY have low Arterial Inflammation. What 62 coHort did in common to accomplish that? Or reduce OXY LDL as Xtronic wants?
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Posted: 5/31/2013 9:16:37 AM
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What did Budoff's 62 coHort eat for such GREAT Results???????


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